Here is how I look at articles like this one.
The article states:
Thus, when we eat these GM crops, we consume NAG. Once the NAG is inside our digestive system, some of it may be re-transformed back into the toxic herbicide. In rats fed NAG, for example, 10% of it was converted back to glufosinate by the time it was excreted in the feces. Another rat study found a 1% conversion. And with goats, more than one-third of what was excreted had turned into glufosinate.
The critical thinking questions to ask here are: What is the
concentration of the herbicide? If 10% is reconverted to the toxic form, how does this correspond to the toxic dose of the compound? Since, by definition, the herbicide is being "excreted in the feces" and since the compound is alleged to be a
neurotoxin where is the evidence that it 1) is absorbed into the bloodstream, 2) passes through the blood brain barrier.
There is some evocation of the matter, in the evocation of
goats but specifically neurological toxicity is not shown. The limits of detection of many molecules is well below the toxicity level. For instance, carbon monoxide is a known toxic compound that
can result in neurologically toxic effects. It is
detectable in almost every human being on earth. Ditto mercury. However the number of instances in which the effects have physiological meaning are relatively
small compared to the population in which these toxins can be shown to have detectable concentrations.
Next we have this grammatically questionable statement, which may or may not have a misplaced modifier.
Glufosinate ammonium is structurally similar to a natural amino acid called glutamic acid, which can stimulate the central nervous system and, in excess levels, cause the death of nerve cells in the brain.
Is the implication here that the amino acid glutamic acid
can stimulate the nervous system and
can cause the death of nerve cells
identical to the implication that glufosinate does the same thing? Glutamic acid is an essential compound in all living things, being one of the 20 basic amino acids. What exactly is this "excess" level?
Next we have this statement:
Perhaps a more critical question may be whether infants or fetuses are impacted with smaller doses. A January 2006 report issued by the Environmental Protection Agency's (EPA) Office of Inspector General said that studies demonstrate that certain pesticides easily enter the brain of young children and fetuses, and can destroy cells. That same report, however, stated that the EPA lacks standard evaluation protocols for measuring the toxicity of pesticides on developing nervous systems. Scientists at the agency also charged that "risk assessments cannot state with confidence the degree to which any exposure of a fetus, infant or child to a pesticide will or will not adversely affect their neurological development."
This is my favorite kind of scientifically misleading statement, the one that attempts to imply that a
lack of information suggests that something is in fact pernicious, even though the absence of information implies that nothing, good or bad, is known. If you read this statement, it is clearly intended to evoke the impression that glufosinate is toxic to (gasp) fetuses and babies! In fact no such causative effect is shown. The reference is to "some pesticides" and in any case the information is at best third hand. The
Office of the (unidentified) Inspector General says that "studies" (no references to the studies themselves, such as a reference to the published scientific literature) "demonstrate" that "some pesticides" (the relation to glufosinate unstated)
easily enter the brains of infants and
can kill some cells. One needs to know right up
which pesticides and the conditions under which they
can kill cells, and under what conditions the conditional word
can is substituted by the word "does" and whether such cell death has any clinical significance. By implication however,
no substance that cannot be proved to
never cause such effects can be acceptable, since the author wishes to imply that the
negative result can
only be proved by testing all compounds on babies, something which clearly will
not happen for ethical reasons for all compounds.
Then there is this statement:
If the herbicide is regenerated inside our gut, since it is an antibiotic, it will likely kill gut bacteria. Gut microorganisms are crucial for health. They not only provide essential metabolites like certain vitamins and short fatty acids, but also help the break down and absorption of food and protect against pathogens. Disrupting the balance of gut bacteria can cause a wide range of problems. According to molecular geneticist Ricarda Steinbrecher, "the data obtained strongly suggest that the balance of gut bacteria will be affected" by the conversion of NAG to glufosinate.
By logical implication here penicillin and amoxicillin are deadly toxic compounds that should be banned. This of course would create situations like that my own mother experienced, being orphaned at 11 when
her mother, my grandmother, died from a common strep infection. This is a classic in poor risk/benefit analysis.
I have only selected a
few samples from this article that use these types of highly questionable associations.
This article is typical of the type that is used to evoke knee jerk luddite reactions to imply that technology must be uniformly rejected. Do I know that glufosinate is a good thing or a bad thing? No, I don't. Is there some risk here? Maybe. Is a nefarious effect proved? No. Far from it. This article should pass for a high school level quiz in the exercise of precisely what is no longer taught in schools:
Critical thinking.
Speaking only for myself, I live on a planet that is dangerously over-populated and which hovers at the brisk of catastrophe of unimaginable proportions. I am decidedly unhappy that this state of affairs has been obtained, but I somehow cling to the hope that the matter can be resolved without resort to mass starvation through the immediate collapse of agriculture, which in these times is
necessarily industrial, at least for the 95th percentile poorest part of the population. In recognizing this, I do not seek some kind of nebulous zero-risk nirvana in which all inhabitants are acutely aware of the "naturalness" of every detectable molecule in their bodies. This does not mean I endorse a cowboy approach to pesticides in particular or chemicals in general, but on the other hand, I do not endorse rote paranoia either. The health food store fantasy for my money is, under the circumstances, a bourgeois conceit that is entirely too self-absorbed for my tastes. I certainly do not approve of the inferential mode of
specious thinking with which this article oozes.
The matter of this compound is, in my view,
not earth shaking. I'm not going to lose any sleep whatsoever worrying about the potential toxicity of glufosinate in my canola oil or my corn oil. In any case, I am routinely informed on this forum, that all of my canola and corn oil will ultimately end up fueling diesel powered Volkswagen Jettas. Almost all of my corn will end up as ethanol for fueling flex fuel Buick Le Sabres. The combustion, I hope, will detoxify any residual glufosinate.