Source:
News release from Michigan State UEAST LANSING, Mich. — A survey, commissioned by the American Association of Veterinary Laboratory Diagnosticians and designed and implemented by Michigan State University toxicologists, has found that more than 300 dogs and cats may have died earlier this year as a result of eating contaminated pet food.
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“Separately, those two compounds are pretty harmless,” said Wilson Rumbeiha, an associate professor in MSU’s Diagnostic Center for Population and Animal Health. “But when combined, they form crystals which can block the kidneys.
“And, unfortunately, these crystals don’t dissolve easily. They go away slowly, if at all, so there is the potential for chronic toxicity.”
It was earlier this year that Rumbeiha and colleagues designed a questionnaire to determine how many animals had become ill or died from eating bad food. The contamination led to a massive recall of tainted pet food.
Collecting data from veterinarians, veterinary technicians and pathologists from April 5 through June 6, Rumbeiha found that 347 cases met the criteria for what he called “pet food-induced nephrotoxicity.” The cases involved 235 cats and 112 dogs.
“Two-thirds of the animals affected were cats, but proportionally, more dogs died from it than cats,” Rumbeiha said.
He theorized that more cats got sick than dogs because of their smaller size. He also found that smaller-breed dogs were more susceptible.
Among some other findings:
* Nearly 98 percent of the 347 cases reported were in the United States, with the other 2 percent from Canada.
* In the United States, Texas recorded the most cases, followed by Illinois and Michigan.
* An equal number of male and female animals were affected.
* The average age of an affected animal was 8 years. The ages ranged from two months to 18 years.
* About 25 percent of the affected animals had a pre-existing condition that made them more susceptible. Most of the conditions were kidney or cardiovascular disease.
Read more:
http://www.newsroom.msu.edu/site/indexer/3263/content.htm